Stroke and Neurological Disorders

Welcome Message

Greetings from the 13th International Conference on Stroke and Neurological Disorders.

We are delighted to welcome you to this prestigious global gathering, scheduled to take place on March 30–31, 2026, in Dubai, UAE, under the theme “Advances in Diagnostics for Stroke and Neurological Disorders.”

This conference will bring together an outstanding community of renowned speakers, distinguished delegates, clinicians, researchers, academicians, industry experts, and healthcare innovators from around the world. Participants will engage in insightful keynote sessions, thought-provoking panel discussions, interactive workshops, and innovative research presentations focused on the latest diagnostic advancements, therapeutic breakthroughs, and clinical approaches in stroke and neurological care.

In addition to keynote and invited talks, the conference offers oral and poster presentation opportunities, giving researchers, young scientists, and students a global platform to showcase their work. These sessions encourage knowledge exchange, academic recognition, and meaningful discussions that contribute to advancements in the field.

Our esteemed speakers will share their expertise and highlight transformative developments that are shaping the future of neurology. Delegates will have opportunities to network, collaborate, and exchange ideas, fostering professional growth and inspiring meaningful progress in neurological research and patient care.

We look forward to your active participation in making this conference an enriching and impactful experience for the entire neuroscience community.

Regards,
Organizing Committee
Stroke Summit 2026
March 30–31, 2026 | Dubai, UAE

About the Conference

The 13th International Conference on Stroke and Neurological Disorders will be held on March 30–31, 2026, in Dubai, UAE. This global meeting is designed to bring together neurologists, stroke specialists, researchers, clinicians, academicians, industry leaders, and healthcare professionals to discuss the latest progress in the diagnosis and management of stroke and neurological conditions. The conference theme, “Advances in Diagnostics for Stroke and Neurological Disorders,” reflects the growing importance of early detection, precision techniques, and innovative approaches that improve patient outcomes.

The event will feature keynote lectures, scientific presentations, panel discussions, and interactive sessions covering a wide range of topics related to stroke prevention, neuroimaging, neurorehabilitation, novel diagnostic tools, and emerging therapeutic strategies. Participants will also have the opportunity to present their research through oral and poster sessions, providing a valuable platform for global visibility and academic recognition.

The conference aims to encourage collaboration, promote research sharing, and support professional development across the neuroscience community. By connecting experts from different regions and specialties, the event fosters meaningful dialogue and paves the way for innovation in neurological healthcare.

Why to Attend

Attending the 13th International Conference on Stroke and Neurological Disorders is an essential opportunity for professionals in neurology to stay at the forefront of the latest advancements. This conference offers cutting-edge insights into new diagnostic technologies, therapeutic strategies, and innovative approaches in stroke and neurological care. Participants will have the chance to:

• Learn from leading experts in stroke and neurological disorders
• Engage in hands-on workshops and interactive scientific sessions
• Stay updated on the latest research, case studies, and clinical breakthroughs
• Earn CPD credits to support ongoing professional growth
• Network with clinicians, researchers, academicians, and global thought leaders
• Foster new collaborations, research partnerships, and career opportunities

Target Audience

The 13th International Conference on Stroke and Neurological Disorders is designed for a wide range of professionals dedicated to advancing neurological healthcare. The event welcomes:

• Neurologists and stroke specialists
• Neurosurgeons and interventional neurologists
• Neuroscience researchers and scientists
• Radiologists and neuroimaging experts
• Rehabilitation specialists and physiotherapists
• Emergency medicine physicians and critical care professionals
• Medical practitioners and clinical researchers
• Academic professionals, professors, and students in neuroscience
• Pharmaceutical, biotech, and medical device industry representatives
• Hospital administrators and healthcare policymakers
• Nurses and allied health professionals involved in neurological care

Sessions and Tracks

Track 01. Stroke Diagnosis

Stroke diagnosis involves rapid identification of neurological deficits through clinical examination, imaging assessment, and physiological evaluation. Early detection relies on recognition of vascular obstruction, cerebral ischemia, and perfusion abnormalities. Diagnostic accuracy is enhanced by neuroimaging markers, biochemical indicators, and standardized stroke scales that classify severity. Timely identification of ischemic changes and hemorrhagic events improves therapeutic decision-making and minimizes neuronal damage. The field continues to advance with improved imaging resolution, faster detection tools, and integrated diagnostic algorithms that support precision-based neurological care.

Track 02. Neuroimaging Techniques

Neuroimaging plays a central role in visualizing brain structure, vascular flow dynamics, and tissue integrity. Techniques such as magnetic resonance imaging, computed tomography, positron emission tomography, and diffusion-weighted imaging provide detailed anatomical and functional insights. These modalities differentiate between ischemic and hemorrhagic pathology, identify vascular occlusion, and detect neuronal injury patterns. High-resolution imaging supports treatment planning, surgical guidance, and monitoring of disease progression. Innovations continue to enhance real-time visualization, perfusion mapping, and advanced brain connectivity analysis.

Track 03. Acute Stroke Care

Acute stroke care focuses on rapid restoration of cerebral blood flow and stabilization of neurological function. Emergency interventions include thrombolytic therapy, endovascular thrombectomy, and advanced airway or circulatory support. Efficient triage, fast imaging acquisition, and precise timing of therapy are essential for reducing infarct size and preserving neuronal tissue. Acute management protocols emphasize hemodynamic stabilization, intracranial pressure regulation, and prevention of secondary brain injury. Advancements continue to refine emergency pathways and neuroprotective strategies.

Track 04. Ischemic Stroke

Ischemic stroke results from obstruction of cerebral arteries leading to reduced perfusion and oxygen deprivation. Pathophysiology involves thrombus formation, embolic occlusion, cellular energy failure, and excitotoxicity-induced neuronal death. Management strategies focus on clot dissolution, vascular recanalization, antithrombotic therapy, and prevention of recurrent cerebrovascular events. Ongoing research explores neuroprotective compounds, advanced perfusion imaging, and molecular targets that mitigate ischemic cascade damage. Understanding vascular risk factors and endothelial dysfunction remains central to long-term prevention.

Track 05. Hemorrhagic Stroke

Hemorrhagic stroke is characterized by intracerebral or subarachnoid bleeding caused by vessel rupture, hypertension, or vascular malformations. The sudden release of blood leads to increased intracranial pressure, cerebral edema, and mechanical compression of neural tissue. Management involves hemostatic stabilization, surgical hematoma evacuation, and monitoring of neurological deterioration. Advances in neurocritical care focus on blood pressure control, vascular repair, and reduction of secondary injury mechanisms. Research continues on biomarkers that predict bleeding severity and outcomes.

Track 06. Neurovascular Health

Neurovascular health encompasses the integrity of cerebral vessels, regulation of cerebral blood flow, and protection of neural tissue from vascular injury. Conditions such as aneurysms, arterial stenosis, and arteriovenous malformations disrupt normal hemodynamics and elevate stroke risk. Diagnostic evaluation includes angiography, ultrasound flow studies, and perfusion imaging. Therapeutic approaches involve endovascular repair, surgical correction, and medical management of vascular dysfunction. Emphasis is placed on endothelial biology, vascular remodeling, and long-term prevention of cerebrovascular events.

Track 07. Neurodegenerative Disorders

Neurodegenerative disorders involve progressive loss of neurons and synaptic connections associated with conditions such as Alzheimer’s disease, Parkinson’s disease, and related dementias. Pathological mechanisms include protein misfolding, accumulation of toxic aggregates, mitochondrial dysfunction, oxidative stress, and neuroinflammation. Diagnostic methods incorporate biomarkers, neuroimaging, and cognitive assessment tools. Treatment efforts aim to slow neurodegeneration through targeted therapies, neurotransmitter modulation, and strategies that protect neuronal circuits. Ongoing research explores genetic influences, molecular pathways, and early detection biomarkers.

Track 08. Neuromuscular and Movement Disorders

Neuromuscular and movement disorders result from dysfunction of peripheral nerves, neuromuscular junctions, or central motor pathways. Conditions such as multiple sclerosis, dystonia, and muscular dystrophy involve impaired signaling, demyelination, or muscle fiber degeneration. Diagnostic evaluation focuses on electrophysiological testing, imaging studies, and molecular genetic analysis. Management strategies include pharmacological modulation, neurorehabilitation, and supportive motor therapies. Research continues to expand understanding of immune-mediated mechanisms, neurotransmission disturbances, and neuroplasticity.

Track 09. Neuroinflammation

Neuroinflammation involves activation of the central nervous system immune response driven by microglial activation, cytokine release, and disruption of the blood–brain barrier. It plays a significant role in stroke, traumatic brain injury, demyelinating diseases, and autoimmune neurological conditions. Molecular pathways include inflammatory cascades, oxidative stress mechanisms, and immune-mediated neuronal injury. Therapeutic strategies target cytokine modulation, immune regulation, and reduction of chronic inflammatory signaling. Advancements continue to explore biomarkers that track inflammatory activity and therapeutic response.

Track 10. Pediatric Neurology

Pediatric neurology focuses on neurological development and disorders affecting infants and children, including congenital abnormalities, epilepsy, metabolic brain diseases, and developmental delays. Pathophysiological mechanisms may involve genetic mutations, abnormal neural maturation, structural malformations, or metabolic dysfunction. Diagnostic assessment uses neuroimaging, electrophysiology, and developmental evaluations to identify early abnormalities. Treatment emphasizes neurodevelopmental support, targeted therapy, and long-term neurological monitoring. Research advances continue to improve understanding of childhood brain plasticity and early-intervention strategies.

Track 11. Technological Innovations in Neuroscience

Technological innovations in neuroscience explore how advanced tools help scientists understand the brain with higher accuracy. Modern imaging methods allow researchers to observe brain activity, structural changes, cellular responses, and connections between neurons in real time. Continuous development in microscopy, computational modeling, and electrophysiological recording supports the examination of nerve cells, synapses, and neural pathways in both healthy and diseased states. Robotic systems, brain–machine interfaces, wearable neural monitoring devices, and automated analytical platforms contribute to improved understanding of neurological functions. These technologies provide deeper insight into sensory processing, learning patterns, memory formation, neural communication pathways, and neurochemical signaling. Artificial neural systems, advanced simulation models, and real-time monitoring technologies help identify abnormalities in neural circuits and support improved interpretation of neurological disorders. These innovations are transforming the study of neuronal structures, behavioral responses, and cognitive functions.

Track 12. Molecular Mechanisms of Neurodegeneration

Molecular mechanisms of neurodegeneration examine how changes inside nerve cells lead to gradual loss of their structure and function. Abnormal accumulation of misfolded proteins, oxidative stress, mitochondrial dysfunction, and impaired cellular clearance processes contribute to neuronal damage. Altered neurotransmitter production, disrupted synaptic communication, and abnormalities in energy regulation also influence the progression of degenerative changes. Inflammatory responses within the central nervous system affect the survival and stability of neurons, while genetic mutations can interfere with normal protein folding and transport. Molecular pathways involving protein aggregation, cellular stress responses, and impaired autophagy play a vital role in the decline of neuronal health. These mechanisms collectively influence memory decline, reduced cognitive abilities, and impaired motor functions. Understanding these molecular changes supports improved exploration of the processes that affect long-term neuronal stability.

Track 13. Neural Stem Cell Biology

Neural stem cell biology focuses on how specialized cells in the brain generate new neurons, glial cells, and supporting structures essential for brain development and repair. These stem cells possess the ability to self-renew and differentiate into multiple neurological cell types, making them crucial for maintaining neural tissue. Cellular signaling pathways regulate their proliferation, migration, and maturation, ensuring proper development of neural circuits. Growth factors, extracellular matrix components, and chemical signals influence the behavior of these cells during embryonic development and adult brain repair. Neural stem cell research examines how these cells respond to injury, inflammation, and degenerative changes. Their capacity to regenerate damaged tissue and restore neural connectivity provides deeper understanding of brain plasticity and developmental patterns. The biology of these cells also highlights the influence of genetics, environmental factors, and molecular regulators on brain formation.

Track 14. Synaptic Transmission and Plasticity

Synaptic transmission and plasticity explain how nerve cells communicate and adapt to new information. Electrical signals travel along neurons and convert into chemical messengers that cross the synapse to activate receptors on neighboring cells. This communication controls memory formation, learning processes, sensory responses, and motor coordination. Synaptic plasticity allows connections between neurons to strengthen or weaken based on repetitive activity, environmental input, and internal chemical signals. Long-term potentiation and long-term depression modify the efficiency of synaptic communication and influence the formation of stable neural networks. Molecular changes within the synapse, including receptor trafficking, protein synthesis, and neurotransmitter regulation, play a central role in adaptation. These processes support the development of complex behaviors, cognitive flexibility, and stable neural circuitry.

Track 15. Neuroinflammation and Immune Response

Neuroinflammation and immune response describe how the central nervous system reacts to harmful stimuli such as injury, infection, toxins, or degenerative processes. Microglial cells act as primary immune regulators within the brain and become activated when they detect cellular damage or abnormal protein accumulation. Astrocytes contribute to inflammatory signaling by releasing chemical mediators that influence neural survival and tissue repair. Prolonged activation of inflammatory pathways can alter synaptic communication, affect neurotransmitter balance, and disrupt normal neuronal function. Cytokines, chemokines, and reactive oxygen species interact with neural structures and influence the progression of neurological disorders. The immune response also affects the stability of the blood brain barrier, allowing additional molecules to enter or exit the brain tissue during stress. This complex interaction between neural and immune systems shapes the long-term outcome of many brain conditions and contributes to both protective and harmful changes within neural networks.

Track 16. Neuroendocrine Regulation

Neuroendocrine regulation examines how the brain communicates with endocrine glands to maintain internal balance through hormone release. The hypothalamus and pituitary gland serve as central coordinators, receiving neural signals and converting them into hormonal instructions that control growth, metabolism, reproduction, and stress responses. Hormones circulate through the bloodstream and influence the activity of organs, tissues, and neural circuits. Changes in hormone levels can alter behavior, emotional processing, memory formation, and energy regulation. The interaction between neurotransmitters and hormonal pathways shapes the body’s response to environmental stimuli and internal physiological demands. Neuroendocrine mechanisms also play a significant role in sleep regulation, appetite, and developmental transitions. Disturbances in these pathways can influence cognitive functions, mood stability, and long-term neurological health.

Track 17. Neural Circuit Development

Neural circuit development explores how networks of neurons form, connect, and refine their communication pathways throughout growth and maturation. During early development, neurons extend axons and dendrites guided by chemical signals, cell adhesion molecules, and environmental cues that determine proper direction and target location. Synapses form when neuronal connections are established and begin to transmit electrical and chemical signals. As the brain matures, neural circuits undergo elimination of weaker connections and strengthening of frequently used pathways, shaping sensory perception, movement control, and cognitive abilities. Genetic factors, molecular signaling proteins, and external experiences influence the pattern of circuitry formation. Development continues through adolescence and adulthood as circuits adapt to learning processes, environmental conditions, and neural activity patterns. Abnormalities in circuit development can affect communication between brain regions and contribute to various neurological conditions.

Track 18. Neuronal Metabolism and Energy Regulation

Neuronal metabolism and energy regulation focus on how nerve cells generate and utilize energy to support their high activity demands. Neurons depend heavily on glucose metabolism and mitochondrial function to produce the energy required for synaptic transmission, ion transport, and structural maintenance. Disruption in metabolic pathways can impair neurotransmitter synthesis, reduce electrical signaling efficiency, and alter neuronal survival. Oxygen availability, blood flow, and nutrient transport through the blood brain barrier influence energy balance within neural tissue. Metabolic stress triggers adaptive responses involving antioxidant defenses, molecular repair systems, and changes in cellular communication. Energy regulation also plays a role in brain development, aging, and responses to injury. Understanding these processes helps explain how metabolic disturbances contribute to neurodegenerative changes and cognitive decline.

Track 19. Neural Communication Pathways

Neural communication pathways refer to the routes through which electrical and chemical signals travel across the nervous system. These pathways transmit information from sensory organs to processing centers and from the brain to muscles, glands, and internal organs. Axonal conduction enables rapid movement of electrical impulses, while neurotransmitters ensure precise communication at synapses. Different pathways control voluntary movement, autonomic functions, sensory interpretation, emotional responses, and cognitive processing. The organization of pathways determines the flow of information between regions such as the cortex, brainstem, hypothalamus, and spinal cord. Damage or disruption to these pathways affects coordination, reflexes, memory, and behavior. Structural and functional mapping of neural circuits helps reveal how distinct pathways contribute to complex processes such as decision making, learning, and adaptive behavior.

Track 20. Neurovascular Coupling

Neurovascular coupling describes how neural activity and blood flow work together to maintain oxygen and nutrient delivery to active regions of the brain. When neurons increase their firing, surrounding cells release signaling molecules that instruct nearby blood vessels to widen. Astrocytes form an important link between neural activity and vascular response by transmitting chemical signals to smooth muscle cells on vessel walls. This coordinated process ensures that active brain regions receive adequate glucose and oxygen to sustain metabolism. Disruption of neurovascular coupling can reduce tissue perfusion, alter synaptic communication, and impair cognitive functions. Changes in vascular reactivity also influence waste clearance and contribute to metabolic stress within neural tissue. Abnormalities in this mechanism are associated with stroke progression, neurodegeneration, and age-related brain changes.

Track 21. Blood Brain Barrier Integrity

Blood brain barrier integrity focuses on the selective barrier that protects neural tissue from toxins, pathogens, and fluctuations in blood composition. The barrier is formed by tightly connected endothelial cells supported by astrocytes and pericytes, creating a highly controlled interface between the bloodstream and the brain. Its transport systems regulate the movement of nutrients, ions, and metabolic products while restricting harmful substances. When the barrier becomes compromised, inflammatory molecules, proteins, and immune cells can enter the brain and disrupt cellular balance. Barrier dysfunction influences neuronal excitability, oxidative stress, and synaptic stability. This structural and functional decline plays a major role in stroke pathology, neuroinflammation, and chronic neurological disorders. Restoring barrier stability is a key focus in many therapeutic approaches.

Track 22. Synaptic Plasticity

Synaptic plasticity explains how synapses strengthen or weaken over time in response to patterns of activity. Synaptic signals are modified through changes in receptor density, neurotransmitter release, and structural remodeling of dendritic spines. Long term potentiation enhances signal transmission between frequently active neurons, while long term depression reduces signaling in less active connections. These processes shape learning, memory formation, and adaptive behavior. Molecular pathways involving calcium ions, protein kinases, and gene expression regulate the intensity and duration of plastic changes. Environmental experiences, sensory input, and developmental factors influence the extent to which plasticity occurs. Disturbances in synaptic plasticity affect cognitive processing, motor coordination, and emotional regulation.

Track 23. Motor Control Pathways

Motor control pathways involve the neural circuits that coordinate voluntary movement, posture, and muscle tone. Signals originate in motor regions of the cerebral cortex and travel through descending tracts that connect to the brainstem and spinal cord. These pathways integrate sensory feedback, balance information, and fine motor adjustments to generate smooth and accurate movements. The basal ganglia and cerebellum play essential roles in refining motor commands and preventing excessive or inappropriate activity. Neurotransmitters such as dopamine, glutamate, and gamma aminobutyric acid regulate communication within these circuits. Damage to motor pathways affects coordination, gait, reflexes, and muscle strength. Understanding these pathways helps explain the biological basis of movement disorders and motor impairment after neurological injury.

Track 24. Cognitive Processing Networks

Cognitive processing networks describe the interconnected regions of the brain responsible for memory, attention, language, reasoning, and decision making. These networks rely on coordinated activity across the cortex, hippocampus, thalamus, and association areas. Neural communication within these networks is influenced by electrical oscillations, synaptic strength, and neurotransmitter balance. Cognitive functions depend on efficient information transfer between multiple circuits that integrate sensory input, emotional responses, and learned experiences. Structural or functional disruptions within these networks contribute to memory deficits, impaired judgment, and reduced problem solving abilities. Biological changes involving inflammation, metabolic stress, and neuronal loss can affect network connectivity and processing speed.

Track 25. Artificial Intelligence in Neurodiagnostics

Artificial intelligence in neurodiagnostics describes the use of computational models that examine structural and functional brain data to detect subtle abnormalities. These systems analyze large sets of neuroimaging results, including magnetic resonance imaging and computed tomography scans, to identify early markers of stroke, microvascular injury, or degenerative changes that may not be visible through standard observation. Algorithms interpret tissue density, blood flow patterns, lesion boundaries, and neural connectivity to support accurate identification of pathological processes. Artificial intelligence tools examine patient specific variables such as perfusion rates, metabolic changes, and neuronal loss patterns to predict progression of neurological disorders. These systems also help classify different types of stroke by learning from previously recorded biological data. Through continuous processing of imaging signals, artificial intelligence contributes to better understanding of structural alterations within the nervous system.

Track 26. Machine Learning in Neural Pattern Recognition

Machine learning in neural pattern recognition focuses on computational techniques that study repeated signals and activity rhythms within the brain. These models analyze electrical patterns from electroencephalography, assess fluctuations in neuronal firing, and detect disruptions in communication between brain regions. By processing large quantities of electrophysiological data, machine learning identifies relationships between abnormal signal waves and specific neurological events such as ischemia, seizure activity, or tissue inflammation. The models learn how neuronal clusters respond to metabolic stress, oxidative imbalance, or impaired blood flow. They also interpret connectivity maps and transmission speed changes to understand how neural circuits reorganize after injury. This approach reveals hidden biological trends that influence disease development and recovery.

Track 27. Intelligent Systems in Stroke Prediction

Intelligent systems in stroke prediction examine biological indicators that signal increased vulnerability within the cerebrovascular network. These systems analyze blood flow irregularities, vessel wall stress patterns, clot formation probability, and metabolic fluctuations within neural tissue. Predictive models integrate biomarkers such as inflammatory proteins, lipid levels, oxidative stress markers, and vascular stiffness measurements to estimate the likelihood of cerebrovascular events. They interpret early signs of impaired neurovascular coupling, endothelial barrier weakening, and regional perfusion changes. Through data-driven assessment, intelligent systems reveal how small disruptions in vascular function progress toward ischemic or hemorrhagic events. This helps deepen understanding of the biological timeline leading to tissue damage.

Track 28. Computational Models of Brain Connectivity

Computational models of brain connectivity explore how different regions of the nervous system interact through structural and functional pathways. These models study cortical thickness, fiber tract density, synaptic strength variations, and oscillatory signaling patterns to build network maps. By examining subtle delays in communication and irregular flow of electrical impulses, computational tools detect alterations associated with stroke, neuroinflammation, or degenerative processes. They reveal how damage to a single pathway influences multiple interconnected circuits and affects cognition, memory, and motor control. These models help illustrate compensatory mechanisms that arise when neurons reorganize or recruit alternate pathways. Through mathematical simulation, connectivity models provide a deeper understanding of how biological networks adapt to injury and disease.

Market Analysis Report

The global market for stroke and neurological disorder diagnostics is expanding steadily as disease prevalence increases worldwide. Recent estimates place the neurological diagnostics market at around 17.5 billion USD in 2023, with projections reaching approximately 23 billion USD by 2030. This growth reflects rising demand for early detection technologies, increasing stroke incidence, and greater investment in advanced imaging infrastructure across hospitals and specialty centers. Strengthening disease surveillance systems and improving access to neuroimaging are further supporting market expansion.

Technological advancements are becoming the primary drivers of future market growth. Artificial intelligence–integrated imaging tools, automated lesion-detection systems, and high-resolution magnetic resonance imaging platforms are reshaping diagnostic workflows. Forecasts indicate that segments involving digital neurodiagnostics and machine learning–based assessment tools may grow at 7 to 8 percent CAGR through 2031, supported by higher adoption of computational decision-support systems. Similarly, therapeutic markets focused on minimally invasive interventions, thrombolytic innovations, and neuroprotective agents are projected to grow at 4 to 5 percent CAGR over the next decade, driven by increasing clinical preference for rapid and targeted treatment approaches.

The rehabilitation and long-term recovery sector is emerging as one of the strongest future growth areas. As stroke survival rates rise, global demand for robotics-assisted therapy, remote monitoring systems, and virtual neurological rehabilitation platforms is increasing. Market projections suggest that the neurorehabilitation technology segment could expand from about 3.8 billion USD in 2025 to more than 7.5 billion USD by 2034, representing a CAGR of nearly 7.7 percent. This upward trend demonstrates the shift toward personalized, technology-enabled recovery models and highlights a strong commercial opportunity across regions such as Asia-Pacific, Europe, and the Middle East.

Participation Benefits 

• Global publicity with 20,000+ online users and 60,000+ abstract views for maximum research visibility.
• Speaker and abstract pages indexed on Google under your name for worldwide recognition.
• Participants can earn up to 15 CME Credits by attending the sessions.
• Opportunity to gain up to 21 CPD Credits certified by the CPD Standards Office, UK.
• All attendees will receive an IOCM Certification.
• Exclusive One-on-One interaction with eminent neurologists and stroke researchers.
• All presentations will be recorded and live-streamed globally.
• Gain insights from keynote talks by world-renowned experts in neurology and stroke science.
• Meet leading industry professionals and decision-makers from neurology-related sectors.
• Engaging symposia and scientific discussions.
• Career development workshops for skill enhancement.
• Pre-conference workshops in major countries for broader participation.
• Best Poster Award and Outstanding Young Researcher Award nominations.
• Special benefits for Group Registrations.

Important Deadlines

Abstract Submission Deadline: February 30, 2026
Early Bird Registration Deadline: January 30, 2026
Normal Registration Deadline: February 28, 2026
Final Registration Deadline: March 30, 2026

Early bird registration is highly recommended to secure your slot and avail discounted rates.
Please submit your abstract before the deadline to ensure timely review and inclusion in the conference program.
Abstract Submission Link
Registration Link

For abstract submission or registration support, you can contact: neurologists@speakermeeting.com

Past Conference Report

Stroke Summit 2025

We are delighted to welcome participants from around the world to the Stroke Summit 2025, proudly hosted by Conference Series in Dubai, UAE, on September 22–23, 2025. This premier event brings together leading experts, researchers, clinicians, and healthcare professionals to share the latest advancements and breakthroughs in stroke research, treatment, and prevention.

The 12th International Conference on Stroke and Neurologicapropel Disorders aims to advance innovative treatments for stroke and various neurological conditions, lling the fields of neurosurgery and neuroscience forward. Guided by the theme "Future Perspectives in Stroke and Brain Health" our mission is to unite distinguished experts in brain health and neurology to share their insights on effective treatment strategies.